创伤基质：多西他赛在休眠乳腺癌逃逸中的作用。
Wounding the stroma: Docetaxel's role in dormant breast cancer escape.
发表日期：2023 Sep
作者：
Tyler T Cooper, Lynne-Marie Postovit
来源：
CYTOKINE & GROWTH FACTOR REVIEWS
摘要：
乳腺癌在休眠期后复发的机制基本上尚不确定。《PLOS Biology》的一项新研究揭示了多西紫杉醇诱导的肿瘤基质细胞损伤刺激细胞因子释放，从而支持乳腺癌细胞的休眠逃逸。版权归©2023年Cooper, Postovit所有。本文为开放获取文章，根据知识共享署名许可协议，可以在任何媒体中自由使用、分发和复制，前提是保留原作者和出处的信息。
The mechanistic underpinnings of breast cancer recurrence following periods of dormancy are largely undetermined. A new study in PLOS Biology reveals that docetaxel-induced injury of tumour stromal cells stimulates the release of cytokines that support dormancy escape of breast cancer cells.Copyright: © 2023 Cooper, Postovit. This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.