肿瘤来源乳酸通过促进组蛋白H3赖氨酸18乳酰化(H3K18la)调控自噬酶RUBCNL表达,从而促进结直肠癌对贝伐单抗的耐药性
Tumor-derived lactate promotes resistance to bevacizumab treatment by facilitating autophagy enhancer protein RUBCNL expression through histone H3 lysine 18 lactylation (H3K18la) in colorectal cancer
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发表日期:2024 Jan
作者:
DOI:10.1080/15548627.2023.2249762
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