皮肤肿瘤在儿童中相对罕见。最常见的是，皮肤癌是由环境因素引起的，特别是紫外线辐射；因此，年龄是发生皮肤癌的最具预测性的因素。然而，出生时患有某些遗传性皮肤病的儿童患恶性肿瘤的可能性明显更大，必须仔细监测和治疗。已发表的大部分数据主要基于白人患者的体征和症状。因此，我们的目的是强调在医学上代表性不足的有色皮肤 (SOC) 患者中这些遗传性皮肤病的皮肤表现和相对差异。我们对 236 篇已发表文章中的 504 名患者进行了文献综述。其中包括针对 17 岁或以下儿童的手稿，其中包含易于理解的病例报告。 SOC 患者的典型表现通常较少，并且疤痕和色素沉着的发生率较高。受影响的患者中近亲结婚的发生率也较高。能够识别非经典体征的医疗服务提供者可以采取适当的管理和治疗方案，从而有可能使 SOC 患者的治疗结果更加符合白人儿童的情况。© 作者 2024。由牛津大学出版社代表英国皮肤科医师协会出版。版权所有。如需商业重复使用，请联系 reprints@oup.com 获取转载和转载的翻译权。所有其他权限都可以通过我们网站文章页面上的权限链接通过我们的 RightsLink 服务获得 - 如需了解更多信息，请联系journals.permissions@oup.com。

Cutaneous neoplasms are relatively rare in children. Most commonly, skin cancers arise through environmental factors, particularly ultraviolet radiation; thus, age is the most predictive factor in developing cutaneous carcinomas. However, children born with certain genodermatoses are significantly more likely to develop malignancies and must carefully be monitored and treated. The preponderance of published data is based mainly on signs and symptoms present in White patients. Therefore, we aim to highlight the cutaneous presentations and relative differences of these genodermatoses among skin-of-color (SOC) patients, who are underrepresented in medicine. We conducted a literature review of 504 patients presented in 236 published articles. Manuscripts with accessible case reports for children aged 17 or younger were included. SOC patients often present with fewer classic findings and have a higher incidence of scarring and dyspigmentation. There is also a higher incidence of consanguinity in affected patients. Providers being able to recognize non-classical signs enable proper management and treatment regimens, potentially bringing SOC patient outcomes more in line with White children.© The Author(s) 2024. Published by Oxford University Press on behalf of British Association of Dermatologists. All rights reserved. For commercial re-use, please contact reprints@oup.com for reprints and translation rights for reprints. All other permissions can be obtained through our RightsLink service via the Permissions link on the article page on our site—for further information please contact journals.permissions@oup.com.